Neuropathy Treatment in Ferguson, MO

A short drive from Ferguson

The clinic at 4477 Woodson Rd, Suite 104 is a short trip via I-170 or Natural Bridge Road, next to Lambert Airport with on-site parking and an accessible office. No long drives, and no excuses to skip a session.

Finding what is behind the burning

Poor circulation, B12 and other nutritional deficiencies, autoimmune conditions and toxic exposures can all drive neuropathy, and the evaluation exists to establish which apply to you. A normal-looking B12 result is a common false reassurance — see why your normal B12 may be lying.

What the program involves

Circulation therapy, regenerative signaling, photobiomodulation, neuromodulation and metabolic correction, in the combination your findings support. Detail: advanced neuropathy treatments, with every plan issued in large print and plain language.

Why the cause matters more than the label

Two people with identical symptoms can have entirely different drivers, and the same drug will fail one and help the other. Correcting the terrain is what makes recovery possible. Reasoning: the science of nerve terrain rehabilitation.

Start free

The Nerve Damage Score is free, takes two minutes, and emails you a plain-language report. Begin here.

Frequently asked questions

Nearby: Florissant · Berkeley · Hazelwood · University City

Who we see from Ferguson

Ferguson is a short drive north of the clinic, and a striking proportion of patients from here arrive with burning or numb feet and no diabetes diagnosis at all. Many have been told their blood sugar is fine. Several will be diagnosed with diabetes within a few years of that reassurance.

That gap — nerve damage present, diabetes not yet diagnosed — is the most important window in this condition, and it is the one most often missed.

Neuropathy that arrives before the diabetes does

Peripheral nerve damage is commonly framed as a complication of diabetes, which implies it comes after. For a substantial group of people it comes first.

Why the fasting number reassures wrongly

Fasting glucose is the last measure to become abnormal. Insulin resistance develops years earlier, and the post-meal glucose excursions that appear to damage small fibers are invisible to a fasting test taken at 8 a.m. A patient can have a normal fasting glucose, a borderline HbA1c, and demonstrable nerve damage simultaneously — and be told nothing is wrong.

What prediabetes does to nerve

Studies of people with impaired glucose tolerance find peripheral neuropathy at rates well above the non-diabetic population. Neuropathy in prediabetes is not a rare curiosity; it is a recognized presentation, and it is the stage at which intervention changes the trajectory most.

What we measure instead

Fasting insulin alongside glucose, a two-hour tolerance test where indicated, and where the picture is unclear a period of continuous monitoring. How normal lab work misses nerve damage sets out why the standard panel is not enough.

Why this window matters more than any treatment

Nerve damage caught at the prediabetic stage is the most reversible form of this condition we see. Damage that has run for a decade under a diagnosed diabetes is not. The single highest-value thing available in neuropathy care is finding it early, and that is an argument for testing properly rather than for any particular therapy.

What treatment looks like at this stage

When nerve damage is caught before diabetes is established, the plan is weighted heavily toward correcting the metabolic environment rather than toward symptom control.

Glucose stability, not just glucose average

Meal composition and timing built around your own measured response, rather than a generic diet sheet. The target is reducing the excursions, which is a different objective from lowering an average.

Nutritional repletion

Deficiencies common in this population are corrected and re-measured. This is inexpensive, frequently overlooked, and one of the few interventions with a fast effect on symptoms.

Direct nerve support

Where indicated, circulation-directed and photobiomodulation therapy alongside the metabolic work. Alpha-lipoic acid has a genuine trial base for symptomatic diabetic neuropathy and is discussed on its merits rather than sold.

The honest framing

This is a condition where timing matters more than technique. Nerve caught early recovers; nerve lost over a decade does not fully return. That is the argument for testing now rather than waiting for a diagnosis to justify it, and it is the whole reason this page exists.

If you already have a diagnosis

Plenty of patients from here arrive with diabetes already diagnosed and neuropathy already established. The window described above has closed, and the objectives change accordingly: halt progression, protect the feet, treat pain without sedation, and address the drivers still doing damage.

That is a smaller ambition than reversal and a realistic one. What determines whether it worsens is largely within reach, which is the part worth acting on.

Cost and access

Coverage is checked before anything is scheduled, and what is likely to be out of pocket is stated in advance rather than discovered afterwards. Insurance and cost questions are answered directly, including where authorization is uncertain.

Visit Regenerve

See the clinic map & directions · Meet Dr. Padda